H-Leu-Ser-Lys-Leu-OH

H-Leu-Ser-Lys-Leu-OH (LSYL), a latency-associated peptide located at the amino terminus of LAP, exhibits an inhibitory effect on TGF-β1 activation. By binding with KRFK, it obstructs TGF-β1 signal transduction, thereby inhibiting the advancement of hepatic damage and fibrosis [1].